Published On: Fri, Mar 20th, 2020

Covid-19 and pneumonic immune system overreaction

The pharmaceutical company Roche is conducting a trial to repurpose an existing drug and gauge its effectiveness against something that is termed in the associated literature and reportage as “Covid-19 pneumonia”. The news is significant because it allows the joining together of two key pieces of data to create a better picture of the nature of the so-called coronavirus disease. Consequently, it appears as if death by Covid-19 could actually be pneumonia brought on by suppression of the immune system by the underlying causes (and their treatment) that are always associated with the deceased in all the cases announced in the UK. Moreover, the “underlying causes” are not as innocuous as they are made to sound, but in fact such major illnesses as cancer and heart disease. Indeed, the death may not have anything or little to do with the presence of a coronavirus in the host at all, meaning that Covid-19, if it exists, would be the minor aspect of the death.

The origin of Covid-19 is in China, and it is very important to keep referring back to this because as the Chinese now deal with their problem so that the world hears no more about the disease in that place, the genesis of a continuing pandemic, which western government wants to use to torment populations in Europe, Britain and North America, is going to be posted into the historical memory hole. At the outset of the Chinese experience, deaths by pneumonia were blamed on a coronavirus (which is a vital point that already gets lost), but there was no certainty about the causation. An intrinsic relationship between SARS-COV-2, as the coronavirus is now called, and pneumonia that was created in China survived conceptually as medical authorities in the west began to deal with Covid-19 (previously called novel coronavirus-infected pneumonia). This was noted in one of the first FBEL articles on the subject:

Since SARS-CoV-2 has been discovered an idea has emerged that it is a killer in its own right. See, for instance, how Wikipedia states that “those affected by the disease are likely to develop a fever and dry cough” – but ultimately notice this: “While the majority of cases result in mild symptoms, some progress to pneumonia and multi-organ failure”.

Given that multi-organ failure can be a complication of pneumonia, what this is ultimately confirming is that Covid-19 is a causation agent of pneumonia.

Generally it appeared to be the case that when supposed Covid-19 killed, it did in the form of pneumonia, aspects of pneumonia (the patient had respiratory problems) or complications of pneumonia (for instance, organ failure).

However, the definitions have shifted, and now it is being said that pneumonia – along with acute respiratory distress syndrome (“aspects”) and kidney failure (“complications”) – is in fact a complication of Covid-19. It should be clear that a trick of semantics has promoted Covid-19 so that it can appear to be deadly without occurrence of pneumonia. Or, in other words, the myth of Covid-19 as being deadly in its own right has been strengthened through a change in language. Presumably, when Roche say they have a drug that can fight “Covid-19 pneumonia”, it doesn’t actually deal with Covid-19, which will be a problem for another drug (and another source of revenue).

But the prognosis for the continuing myth of Covid-19 muscularity is not good. Recently, information has started to come out of Italy that will prove powerful stuff to arm those who would see Government brought to justice for wrecking the economy and blaming it on Covid-19. It has been found that 99.2% of deaths in Italy officially attributed to Covid-19 involved serious pre-existing illness. The Italian Institute of Health, from whence the data comes, cannot be sure that these deaths were not due to the underlying health conditions of the patients; indeed (via Jon Rappoport) the President of the Institute could not be sure that any death was caused by Covid-19.

This information has confirmed the suspicions of those who are watching events with a sceptical eye, and has established a fact: that a varying range of serious ailment is connected with death that has nevertheless been blamed entirely on Covid-19.

One way of reacting to this fact is to insist that there are two common factors in the deaths, the presence of SARS-COV-2 as the root cause, and then a pre-existing illness that has made the host susceptible to the disease caused by the coronavirus, but this would be wrong. Firstly, we cannot be sure if the tests to find the coronavirus are reliable, and we can’t be sure of the presence of the coronavirus in the host just because he presented the so-called symptoms of those “Covid-19 complications” discussed above.

On the other hand, the common factor in the deaths is more likely to be immunosuppression, or the compromising of the immune system so that it cannot respond normally to an infection of the host. This state of affairs can come about by the patient’s ailment and indeed by the medical treatment for it. As such, it makes great sense that the drug being tested by Roche, called Actemra, is one that is used to treat an immune system reaction. Moreover, with this information, we can tie the pneumonia aspect of Covid-19 to a faulty immune system, and then to the ever present pre-existing illnesses.

Actemra is presently used to manage what is known as Cytokine release syndrome (CRS), which is an overreaction of the immune system, described by Wikipedia as follows:

[CRS is] a form of systemic inflammatory response syndrome that can be triggered by a variety of factors such as infections and certain drugs. It occurs when large numbers of white blood cells are activated and release inflammatory cytokines, which in turn activate yet more white blood cells. CRS is also an adverse effect of some monoclonal antibody drugs, as well as adoptive T-cell therapies.

The thing to note is the reference to treatment of cancer. A T-cell is an immune system cell (implying to a layman that the cancer is lymphoma or leukemia). The literature for Actemra states that the drug is used for CRS that occurs during something called CAR-T cancer therapy, which involves the adaptation of T-cells to fight the cancer.

Simply put, this data is telling us that pneumonia (an inflammatory response) can be triggered by cancer treatment. Indeed, Chinese medics used Actemra on their pneumonia patients, but the following paragraph from a report on the drug trial (linked to above) shows how the activity of the Chinese has been conflated with treatment of Covid-19:

China’s National Health Commission added Actemra to its updated COVID-19 treatment guidelines in early March after Chinese physicians on the front line reported positive outcomes from its use to control dangerous lung inflammation in serious and critically ill patients.

That endorsement apparently triggered wide interest in the drug’s potential in COVID-19.

What we should actually probably deduce is the Chinese were indeed treating pneumonic overreaction of the immune system due to cancer treatment rather than a so-called “Covid-19 pneumonia”. So, rather than CRS (“in its more severe form known as cytokine storm”) being “a key hallmark of… [Covid-19] worsening”, as the above mentioned reportage makes the case, it is an indication of treatment for what is always undersold in the west as “underlying causes”. Here’s another example: instead of it being the case that “cytokine storm syndrome…[is] the immune response that’s killing many who die of Covid-19”, it should be said that is indicative of those downplayed “underlying causes” in fact being the chief (and lethal) issue.

On reflection, then, the Chinese were arguably faced with a concentration of population that was overcome by local chronic living conditions, and suffering from pneumonia that was either complication of low grade illness or brought on by compromised immune systems, or the immune suppression in patients who were suffering from serious illnesses, also connected to local conditions.

Ultimately, the truth of the matter is perhaps staring us in the face when we look at the phrase “Covid-19 pneumonia” and analyse the language. The term suggests that the pneumonia is a thing begot by Covid-19 so that we might think of Covid-19 pneumonia either as a complication of low grade illness, like a cold, or as another name for a complication involving a weakened immune system. Either way, it makes the presence of SARS-COV-2 potentially redundant, or at least immaterial in an immune system healthy population. Which brings us to the fundamental issue of whether or not the novel coronavirus is responsible for anything, or if it is merely coincidental – that’s if it exists at all.

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